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that may be, stimulation of CD74 with exogenous MIF may well cause an too much inflammatory point out, as MIF is able to stimulating CXCR2 and CXCR4 receptors In combination with CD74. CXCR2 and CXCR4 receptors when activated market inflow of neutrophils and lymphocytes, respectively (sixty three, 64). MIF-two in distinction to MIF seems to absenc